Malaria eradication and its effect on mortality levels

Abstract It is argued in this article that malaria eradication was only one of a group of factors which were responsible for the lowering of mortality levels in Ceylon in the years after 1946. The magnitude of the contribution made by these other factors has not generally been taken into account since they took effect during the period of malaria eradication, but since some of them were carried out in a few of the endemic malarial areas during the pre-eradication period, some attempt to measure them can be made of their impact on mortality levels. Further the effect of malaria eradication on mortality levels in the absence of these other measures is also studied by examining the case of Guatemala where in spite of malaria eradication the decline in mortality levels has not been so significant as in Ceylon.     

Malaria eradication and its effect on mortality levels: A comment

Abstract In his article 'Malaria eradication and its effect on mortality levels' (Population Studies d21, 3, November 1967) Dr. S. A. Meegama criticised the approach followed by Professor Peter Newman in an earlier investigation of the effects of malaria eradication in Ceylon. In the present series of comments and rejoinders, Professor Newman and Dr. Meegama discuss in detail their interpretations of the data and the methods they have used in assessing the role of malaria eradication.     

The decline in maternal and infant mortality and its relation to malaria eradication

Abstract In his article 'Malaria eradication and its effect on mortality levels' (Population Studies d21, 3, November 1967) Dr. S. A. Meegama criticised the approach followed by Professor Peter Newman in an earlier investigation of the effects of malaria eradication in Ceylon. In the present series of comments and rejoinders, Professor Newman and Dr. Meegama discuss in detail their interpretations of the data and the methods they have used in assessing the r?le of malaria eradication.     

Religious attendance and mortality: Implications for the black-white mortality crossover

This study investigates the relationships among religious attendance, mortality, and the black-white mortality crossover. We build on prior research by examining the link between attendance and mortality while testing whether religious involvement captures an important source of population heterogeneity that contributes to a crossover Using data from the Established Populations for Epidemiologic Studies of the Elderly, we find a strong negative association between attendance and mortality. Our results also show evidence of a racial crossover in mortality rates for both men and women. When religious attendance is modeled in terms of differential frailty, clear gender differences emerge. For women, the effect of attendance is race- and age-dependent, modifying the age at crossover by 10 years. For men, however; the effect of attendance is not related to race and does not alter the crossover pattern. When other health risks are modeled in terms of differential frailty, wefind neither race nor age-related effects. Overall, the results highlight the importance of considering religious attendance when examining racial and gender differences in age-specific mortality rates.     

Models and Proposals for Malaria: A Review

Mathematical models to study the dynamics of malaria continue to be developed and upgraded on the parasite component, which is the causative agent for malaria; on the human component that serves as a reservoir of infection for the blood feeding female mosquitoes; on the disease transmitting vector, the component mostly responsible for the movement of the parasite agent from one human to another; or on the life cycle of the malaria parasite as a pathogen both within the human and vector populations. The consideration of so-far neglected features can be beneficial for the control of malaria.     

Estimating infant and childhood mortality under conditions of changing mortality

Summary It is well known that estimates of infant mortality obtained using Brass's technique are very accurate. Biases are introduced, however, when one or more of the assumptions on which it relies are violated. Departures from the assumption of constant fertility may be handled by using a variant of the technique which depends on information on the age distribution of surviving children, rather than on indexes of the fertility function. Violations of the assumption of constant mortality - an increasingly common situation in most developing societies - produce upward biases in the estimates. The amount of bias is a function of the speed of mortality decline, the characteristics of the fertility pattern and, finally, of the age of the mother. This paper presents a simple technique which corrects these biases, and in addition, generates estimates of the parameters of the mortality trend. It differs from others in that it uses a cohort definition of mortality decline and relies on knowledge of the age structure of surviving children rather than on indexes of the fertility pattern.     

The fall in household size and the rise of the primary individual in the United States

The long-term fall in household size in the United States is discussed within the framework of the aging of the population, continuing as the effects of fertility and mortality decline accumulate. Using distributions of households by size from U.S. census data 1790–1970 and a components of change analysis on primary individuals for 1950–1974, household changes are related to demographic change for the periods 1790–1900, 1900–1950, and 1950–1974. Fertility and mortality declines have unambiguous impact on household size until the increases in primary individuals begin. But these, too, have a theoretically interesting, if indirect relationship to population structure.     

Beyond infant mortality: gender and stillbirth in reproductive mortality before the twentieth century

Though it has been the largest component of reproductive mortality since its statutory registration in 1928, stillbirth has received little attention from historical demographers, who have relied on the more orthodox indicator of early human survival changes - "infant mortality". The exclusion of stillbirth hampers demographic analysis, underestimates progress in newborn vitality, and over-privileges post-natal causes in theoretical explanation. A case is made for estimating stillbirth before 1928 as a ratio of early neonatal death, and for employing perinatal mortality as an historical indicator of female health status. The long-run trend of reproductive mortality (encompassing mature foetal and live born infant death during the first eleven months) reveals a substantial decline in perinatal causes in the first industrial century (1750-1850), implying a major concurrent improvement in the nutritional status of child bearers. Reproductive mortality is a more complete indicator of death in infancy. It offers demographers a means of fracturing the fertility versus mortality dualism and a potential purchase on gender as a demographic variable, while re-opening the case on mortality in the demographic dynamic of the world we have lost.     

Estimates of mortality from infant mortality rates

Estimates of mortality rates and expectation of life at birth, using infant mortality rates, are examined on the basis of 150 life tables for both sexes. Least squares linear estimates are given as well as estimates of their variances. Model life table calculations, as proposed by the U.N. Population Branch, are then compared with these unbiased minimum variance estimates and shown to overestimate the expectation of life by more than two years on the average, and to be at most 68% efficient. Though better estimates are provided in this paper, their variance is still so large as to cast doubt on the practical usefulness of anv estimates based exclusively on infant mortality rates.     

Tobacco smoking and the sex mortality differential

This paper examines the effects of tobacco smoking on the sex mortality differential in the United States. It is found that all forms of smoking combined account for about 47 percent of the female-male difference in ₅₀ e ₃₇ (life expectancy between ages 37 and 87) in 1962,and about 75 percent of the increase in the female-male difference in ₅₀ e ₃₇over the period 1910–62. When these percentage effects of smoking are decomposed each into a sum of contributions by age and immediate medical cause of death, the degenerative diseases acting at the older ages are found to be of primary importance. The above results appear in large part to explain why the degenerative diseases also account for most of the 1910–65 increase in the female-male difference in life expectancy at birth. The analysis assumes that spurious effects due to the correlation of tobacco consumption with other mortality-related factors are small compared to the causal effects of tobacco consumption itself.     

On the theory and measurement of the determinants of mortality

This article develops a model of mortality that shows how biological, demographic, and environmental factors interact to affect an individual's probability of dying. To illustrate the usefulness of the model, we derive from it (as special cases) the Brass system of model life tables and the proportional-hazard mortality model and apply a logit version of the model to analyze the determinants of child mortality in Sri Lanka.     

Reconsidering mortality compression and deceleration: an alternative model of mortality rates

In this research we develop a model of mortality rates that parameterizes mortality deceleration and compression, permits hypothesis tests for change in these parameters over time, and allows for formal gender comparisons. Our model fits mortality data well across all adult ages 20-105 for 1968-1992 U.S. white data, and the results offer some confirmation of findings of mortality research using conventional methods. We find that the age at which mortality deceleration begins is increasing over time, that decompression of mortality is occurring, and that these trends vary substantially across genders, although male and female mortality patterns appear to be converging to some extent.     

Evolution of fertility and mortality

Historical and current fertility trends in both Quebec and Canada as a whole are surveyed. While fertility among French Canadians was higher than that in neighboring provinces until the mid-20th century, in 1968 Quebec's crude birthrate was the lowest in Canada, and in 1972 it was 13.8 vs. 15.9 (the national birthrate). This reversal is explained in terms of the demographic transition theory, the declining influence of organized religion, and new opportunities for social mobility for minority groups. The birthrate throughout Canada is also declining. Although recent cohort studies are incomplete because women have not yet finished their reproductive years, it appears that completed family size will be lower than at any time in Canadian history. The period total fertility rate indicates an average family size of 1.8 children in 1976, but it is unclear whether this represents an actual reduction in family size or the postponement of childbearing. The sharpest fertility decline has been among women aged 35-49, but peak fertility rates have shifted from the 20-24 age group to those aged 25-29. Fertility is negatively related to education, and the lowest fertility is found among the intermediate income groups. Since the 1969 lifting of the ban on contraceptive sales and advertising, family planning activities have been stepped up. Also removed was the total ban on abortion. In 1975 there were 14.9 therapeutic abortions per 100 live births, but it has been charged that abortion standards are being applied inequitably from hospital to hospital.     

Control Strategies for the Spread of Malaria in Humans With Variable Attractiveness

The transmission dynamics of malaria in humans with variable attractiveness is modeled with the possibility of prophylactic measures such as personal protection and treatment. At the disease-free equilibria, the basic reproduction number and a measure for effective disease control are computed. Simulations show that a 10% increase in personal protection leads to a decrease in secondary transmission, and that a 10% increase in the treatment rate leads to a 7% reduction in secondary transmission. Personal protection and effective treatment should be applied together. Personal protection fails with increasing degree of attractiveness.     

Premature birth and the changing composition of newborn infectious disease mortality: Reconsidering “exogenous” mortality

Linked death and birth records from San Antonio, Texas reveal that infectious infant mortality is increasingly a function of premature birth and low birth weight. Between 1935 and 1944, 4% of infectious infant deaths had associated causes involving prematurity and related conditions; by 1980, 25% of infectious infant deaths involved prematurity and more than 40% of those infants weighed less than 2,500 grams. The shift in birth-weight composition results almost entirely from an increase in very low-weight births. Under conditions of advanced perinatal technology, infectious infant mortality should no longer be viewed as wholly exogenous. These findings further undermine the contemporary relevance of the exogenous-endogenous distinction.     

人口死亡研究的展开

当前 ,在人口死亡现象和研究方面有一些新的内容值得注意 :(1 )人口寿命的延长趋势。发达国家的人口寿命增加的幅度虽然很小 ,但还有所增加 ,人口寿命增长是否有极限 ?发展中国家的平均寿命一度增加很快 ,但现在已放慢了速度。今后世界人口的寿命将如何变化 ?(2 )从人口死亡到人口健康的研究。随着人的平均寿命的延长和人口老龄化 ,不健康的人群绝对数和占全部人口的相对比例也增大。人们对生命质量的关心 ,呼唤着对人口健康研究的重视。 (3 )环境对人口健康 ,人口死亡的影响 ,人口、资源、环境问题已成为全球共同关注的问题。由于人们不合理的生产和生活方式导致了自然环境的破坏 ,已危及到人类的自身的生存。环境公害 ,有毒物质对人体的伤害 ,几乎无人能够幸免。 (4 )“安乐死”的问题。在人类历史上 ,自然死亡一直被人们认为是结束生命的唯一合理形式。这个理念现在受到了挑战。关于安乐死是否合理牵涉到现存的人性、伦理、道德和法律等诸多方面 ,要判断是非 ,可说是世界上的一道难题。     

Subjective mortality hazard shocks and the adjustment of consumption expenditures

I estimate the effect of shocks to subjective mortality hazards on consumption expenditures of retired individuals using the Survey of Health, Ageing and Retirement in Europe. I measure mortality expectations with survey responses on survival probabilities. To create plausibly exogenous variation in mortality hazard, I use the death of a sibling as an instrument. My results show that survey responses contain economically relevant information about longevity expectations and confirm the predictions of life-cycle theories about the effect of these expectations on intertemporal choice.