城市外来务工人员的工作和居住环境及其健康效应——以深圳为例

20世纪80年代以来,我国快速的人口流动与事实城市化对城市环境与人口健康提出了严峻的挑战。本研究通过在深圳市开展的调查,分析了城市外来务工人员的工作、生活环境特征与身心健康状况,初步检验了环境因素对外来务工人员身心健康的净效应。研究发现,相对于其他社会经济因素而言,居住环境和工作环境对外来务工人员的健康具有更为突出的影响。改善外来务工人员的工作和居住环境、倡导良好的保健行为和生活习惯,对于改善其健康状况具有重要意义。     

人口内部均衡发展研究——以西部地区为例

本文从量与质两个方面对人口内部均衡发展概念进行界定——在人口发展过程中,人口内部影响因素在发展数量、质量等方面逐步实现均等或协调,而这种均衡是暂时的、相对的,且不断的由低级均衡向高级均衡发展演替。在此基础上从人口发展、人口内部均衡度、人口内部均衡发展梯度、人口内部均衡可持续发展度四个视角构建人口内部均衡发展评价模型,并对西部地区进行实证分析。主要结论:(1)2000~2007年间,西部人口发展、人口内部均衡度及人口内部均衡可持续发展度呈增长态势,其增速高于同期全国水平,且西部各省间发展差距较大;(2)西部地区人口发展水平较低,位于0.48~0.59之间,属由启蒙后期进入过渡前期阶段;(3)西部人口内部均衡度低,位于0.35~0.42之间,属由轻度失调进入濒临失调阶段;(4)西部人口内部均衡可持续发展度较低,位于0.455~0.544之间,属过渡阶段。     

“发展型”或“政策型”生育率下降?——基于翼城“农村二孩”试点的分析

中国生育率在过去30年持续下降至较低水平,围绕下降的主要动因及其政策涵义的争论热烈。文章以"农村二孩"试点之一的翼城为案例,重新梳理和检视了在已有研究中较为忽视的翼城试点出台的背景和政策实施细则,并从多个方面考察了翼城生育政策对生育行为和生育意愿的影响。与已有研究对翼城试点的认识不同,政策文献回顾表明:"农村二孩"生育政策只是在初婚时间、生育子女数和生育间隔方面做出一些技术性"调整"以缓解国情与民情的矛盾,并没有实质性地改变实施计划生育这一基本国策的初衷。多维度数据分析交叉印证了在实施限制性的生育政策以后,社会经济发展变量不能解释生育率的下降,翼城现有生育行为和生育意愿更多是政策干预的结果,是政策促成的外生性转变,而非由于社会经济发展导致低生育意愿的内生性转变。     

A Preliminary Comparison Study of Online and Face-to-Face Counseling: Client Perceptions of Three Factors

This preliminary study examined the similarities and differences between online and face-to-face counseling with regard to general mental health, working alliance, and social presence. Participants included 50 current counseling clients (37 face-to-face, 13 online) who all were recruited through online mediums. Results indicated that online counseling clients perceived a significantly stronger working alliance on the total Working Alliance Inventory-Short Form as well as the Goal subscale than did those who received face-to-face only counseling. No significant differences existed between the groups on general mental health or social presence. These results suggest that a more controlled study with a larger sample might find that a strong working alliance can be developed through online counseling.     

Weapons of penile smooth muscle destruction: Androgen deficiency promotes accumulation of adipocytes in the corpus cavernosum

In men with erectile dysfunction, venous leakage is a common condition among non-responders to medical management and is attributed to penile smooth muscle atrophy. Androgens play a role in regulating trabecular smooth muscle growth and function. Further, androgens stimulate differentiation of progenitor cells into smooth muscle cells and inhibit their differentiation into adipocytes. We postulate that androgens exert a direct effect on penile tissue to maintain erectile function, and that androgen deficiency produces metabolic and structural imbalances in the corpus cavernosum, resulting in venous leakage and erectile dysfunction. To date, research efforts on the mechanisms by which androgens regulate penile erectile physiology have mainly focused on investigating the role of the NO/cGMP pathway. However, androgen-dependent mechanisms that regulate tissue remodeling have been poorly defined. Characterization of the molecular and cellular mechanisms by which androgens regulate corpus cavernosum structural and functional integrity would provide significant gains in knowledge and understanding of an important pathogenic process. In this review, we discuss the potential role of androgen in maintaining differentiation of progenitor cells into smooth muscle lineage and inhibition of differentiation into adipocytes. Androgen deficiency promotes differentiation into adipogenic lineage, and accumulation of adipocytes in the corpus cavernosum may contribute to erectile dysfunction.